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Concomitant administration of memantine with the antispasmodic agents, dantrolene or baclofen, can modify their effects and a dosage adjustment may be necessary.
Colocasia - Elephant Ear esculenta `Black Magic' - Large, heart-shaped dark purple-black leaves. Tuberous, spreading form. Good pond margin, bog, container, landscape plant. To 3-5' Tall X 3' Wide. Zone 9. 1I - #1 Can New! esculenta `Pink China' - Large, heart-shaped dull green leaves with purple blotches. Tuberous, spreading form. Good pond margin, bog, container, landscape plant. To 4-5' Tall X 4-6' Wide. Zone 9. 1I - #1 Can New. Order antispasmodic onlineRhage. Acacetin at 25-100 mg kg oral dose to mice reduced capillary fragility and at 50-100 mg kg it reduced vascular permeability57, 58. Patuletin reduced the capillary permeability and was also reported to have antispasmodic and hypotensive effects. 10. Antimicrobial activity: Flavonoids and esters of phenolic acids were investigated for their antibacterial, antifungal and antiviral activities. All samples were active against the fungal and gram-positive bacterial test strains and most showed antiviral activity59. i ; Antibacterial Activity: Antibacterial activity has been displayed by a number of flavonoids. Twenty-five out of one hundred and eighty two flavonoid studies were found to be active against many bacteria. Most of the flavonones having no sugar moiety showed antimicrobial activities where as none of the flavonols and flavonolignans tested showed inhibitory activity on the microorganisms59 . Antifungal Activity: Number of flavonoids isolated from peel of tangerine orange, when tested for fungistatic activity towards Deuterophoma tracheiphila showed promising activity. Chlorflavonin was the first chlorine-containing flavonoid type antifungal antibiotic produced by strains of Aspergillus candidus60. Antiviral Activity: Flavonoids also displayed antiviral, including anti-HIV activity. It has been found that flavonols are more active than flavones against herpes simplex virus type 1 and the order of importance was galangin kaempferol quercetin61. Recently, a natural plant flavonoid polymer of molecular weight 2100 Daltons was found to have antiviral activity against two strains of type-1 herpes type simplex virus, including a thymidine-kinase deficient strain and type -2 herpes simplex virus62. Out of twenty-eight fiavonoids tested, flavan-3-o1 was more effective than flavones and flavonones in selective inhibition of HIV-1, HIV-2 and similar immunodeficiency virus infections63. Details of the flavonoids and their antimicrobial activity were given in Table 2 and aprepitant. Buy cheap antispasmodicCheap antispasmodic onlineDo read the article by Stephen Jay Gould called The Median Isn't the Message. It can be found on the World Wide Web at : cancerguide , Steve Dunn's very helpful Cancer Guide. The article explains how to look at the prognosis statistics and provides positive reinforcement about what may lie ahead. Leukemia was first described only about 150 years ago. Since that time there have been substantial advances in diagnosis, tracking of cases, classification although that is constantly evolving as diagnostic tools have become increasingly sophisticated ; , and treatment. The developmental, physical, environmental, and societal factors that may cause leukemia are not yet known. According to the present literature, it is most likely that the principal causes of leukemia have yet to be discovered. The following patient reconstructs a conversation she had with her doctor, in which she gleaned causative information without even asking for it. When I was diagnosed with CLL 10 years or so ago, my doctor said to me, "Well you know, you test positive for having had the Epstein-Barr virus." "That's nice, " I replied. "What is Epstein-Barr, and what does that have to do with lymphocytic leukemia?" "It's a B lymphocyte herpesvirus that starts out like a respiratory infection. But once you've had it, the traces stick around for life. You probably got it when you were a very young child." Then he went on to tell me that it is associated with leukemia, lymphoma, and chronic fatigue syndrome and that most of his leukemia and lymphoma patients showed traces of it in their blood work. I'm a fifty-year-old mother of three, and here we are discussing a childhood disease that I probably had before age ten. Was I supposed to believe this was a cause of my leukemia? The doctor was very careful to explain that it was an indicator that is found in many patients. He made no claim that it was a cause of the disease. By the time he was finished talking, however, I was anxious to research other possible causes of the disease. ANTISPASMODIC AND ANTICHOLINERGIC AGENTS AND PROPULSIVES SYNTHETIC ANTISPASMODIC AND ANTICHOLINERGIC AGENTS Synthetic anticholinergics, esters with tertiary amino group 01 Oxyphencyclimine 20 03 Camylofin 0.2 04 Mebeverine 0.3 05 Trimebutine 06 Rociverine 07 Dicycloverine 08 Dihexyverine 09 Difemerine 18 and aptivus. Kalkstein, L. 1999. Climate-Health showcase projects: International Heat Health watch-warning systems. Proceedings from the Biometeorology and urban climatology at the turn of the Millennium: ICB-ICUC Sydney, 8-12 Nov. 1999. Warnings: Not recommended in primary depressive disorders or psychoses. As with all CNS-acting drugs, warn patients not to operate machinery or motor vehicles, and of diminand other CNS depressants. Dependence: Withdraw symptoms like those noted with bariturates and alcohol have occurred following abrupt discontinuance of benzodiazepines including convulsions, tremor, abdomin and muscle cramps, vomiting and sweating ; . Addiction-prone individuals, e.g. drug addicts and alcoholics, shotdd be under careful surveillance when on benzodiazepines because of their predisposition to habituation and dependence. Withdrawal symptoms have also been reported following abrupt discontinuance of benzodiazepines taken continuously at therapeutic levels for several months and aranesp. Alents and shares the highest amino acid sequence identity to aggrecan. Amphioxus lacks most of the cell and tissue types that we associate with a HA matrix in vertebrates, and it will, therefore, be extremely interesting to localize the various components of the HA-dependent ECM in this organism. It has been hypothesized that two large, genome-wide, duplication events occurred relatively early in vertebrate evolutionary history 29 ; . The alternate hypothesis states that multiple smaller scale duplications have occurred 30 ; , which have had the collective effect of generating many gene families in the higher vertebrates. Our model fits well with either hypothesis. In the origin of the 2PTR subfamily, the first duplication event appears to have been followed by specialization of one of the gene pairs, such that its expression became restricted to neuronal cells. We propose that this event happened in an early vertebrate. The second gene duplication event generated the four paralogous HAPLN-CSPG gene clusters, as we know them. It is highly likely that a more recent rearrangement event moved HAPLN2 relative to brevican. This gene organization is observed in both human and mouse. We propose that two similar genome-wide duplication events resulted in the higher vertebrate 1PTR subfamily, although establishment of the paralogous relationships between CD44, LYVE1, and KIAA0257 has not been possible to establish at this point. We propose that the first genome-wide duplication event yielded the CD44 and stabilin branches. The second event generated CD44 and LYVE1 or KIAA0257 and TSG-6 and a STAB gene. More recent duplication events are proposed to have resulted in the two STAB genes and the CD44 and KIAA0527 genes. KIAA0527 may encode a novel cell-surface HA receptor, al. Date Assessment Initiated: Information Source please circle ; Resident R ; , Family F ; , Chart C ; , RN, RPN, PSW, other. A. RELEVANT MEDICAL & OR SURGICAL CONDITIONS From Resident, Family, Chart ; 1 Immobility Issues 1 Cognitive Problems 1 Arthritis 1 Dementia 1 Other 1 Other 1 Neurological Conditions 1 Genito-Urinary GU ; Problems 1 Stroke 1 Recurrent Urinary Tract Infections 1 Previous G U Surgery or Injury 1 Parkinson's Disease 1 Multiple Sclerosis 1 Prostate Problems 1 Spinal Cord injury 1 Other 1 Other 1 Gastro-Intestinal GI ; Problems 1Medical Conditions 1 Chronic constipation 1 Diabetes 1 Diverticular disease 1 Hypertension 1 Hemorrhoids fissures 1 Hypothyroidism 1 Previous colon surgery 1 Heart Problems 1 Irritable bowel syndrome Weight : kg ; 1 Other MEDICATIONS See over Antacids with aluminum Analgesics NSAIDS Anticholinergic Antispasmodic Anti-emetics Antidepressants Antihistamines Anti-hypertensives Anti-Parkinson agents Anti-psychotics Calcium Channel Blockers Cholinergic Diuretic Histamine-2 blockers Iron supplements Laxatives Narcotic analgesic Sedative hypnotic Other C. URINARY CONTINENCE HISTORY Urinary Urinary Incontinence Incontinence Pattern UI ; Frequency and Timing B. Y N Comments and aredia. Fluid - RTF ; . The different caliber paper points were placed individually in 12 x tubes, which were then labeled and closed with cotton and sterilized in an autoclave at 120C, for 20 min. Bacteriological samples were collected immediately after crown access by introducing 3-4 sequential sterile absorbent paper points, of a size visually compatible with the root canal diameter, 2-3 mm before the radiographic apex or the limit of the physiological root resorption, as determined by diagnostic radiographs. After approximately 1 min, the paper points were removed from each root canal and placed in a test tube 13 x 100 mm ; containing 2.0 ml of reduced transport fluid RTF ; , prepared according to Syed and Loesche19, that is recommended for the maintenance of viable anaerobic microorganisms. The tube was closed hermetically and processed for microbiological analysis. After samples collection, the root canals were treated for the immediate and progressive neutralization of septic toxic content using K-files and copious irrigation suction with 2.5% sodium hypochlorite followed by odontometry 1 mm before the radiographic apex or the limit of the physiological root resorption. Biomechanical preparation was carried out with sequential K-files and irrigation with 2.5% sodium hypochlorite. The canals were subsequently dried with sterile absorbent paper points and filled with EDTA Odahcam Herpo Produtos Dentrios Ltda., Rio de Janeiro, RJ, Brazil ; , and mixed for 3 min with a K-file to remove smear layer. The canals were irrigated and dried, and then filled with calcium hydroxide paste Calen PMCC, S.S. White Artigos Dentrios Ltda., Rio de Janeiro, RJ, Brazil ; with a special syringe ML, S.S. White Artigos Dentrios Ltda., Brazil ; . The pulp chamber was sealed with zinc oxide eugenol cement IRM, Dentsply Indstria e Comrcio Ltda., Petrpolis, RJ, Brazil ; . After 14-30 days, the intracanal dressing was removed and the canals were filled with Calen thickened with zinc oxide, as recommended by Silva and Leonardo15, and restored. Order antispasmodicCENTRAL LOUISIANA STATE HOSPITAL, P.O. Box 81, Pineville, LA, 71360 has an opening for a Psychiatrist IV. Salary begins at , 160 per year with maximum provisions up to , 792 per year. Louisiana license required. On grounds housing available. Forty hour work week. Retirement system available for applicants under age 60. Annual and sick leave credited at the end of each pay period. Hospitalization and Life Insurance offered at minimum cost for employee. Approximately 12 paid holidays per year. Excellent weather year. The Applicant shall furnish to the Insurer any information required for the Insurer to administer the Insurance. The Applicant shall have records available for the Insurer to inspect at any time while insurance is in force, and for up to the earlier of three years after termination date, or final adjustment and settlement of claims is made. The Insurer reserves the right to waive or change any of the above requirements at any time. INSURER UNDERWRITING REQUIREMENTS The Applicant is required to submit an Application for Insurance for self and or for all eligible Dependents to be insured. Insurance for any person is not effective until the date specified by the Insurer. Depending upon the law, the Insurer may have the right to decline the Application for any person for whom information has been submitted in the Application. We will waive the pre-existing limitation for conditions disclosed on this application, but we may place an exclusion rider on certain condition s ; . TERMINATION OF INSURANCE You may terminate insurance at any time by providing us written notice prior to the requested termination date. The termination date will be the first of the month. Insurance will terminate at 12: 01 a.m. Central Standard Time on the termination date. The Insurer will terminate insurance if the Applicant fails to pay premium on the due date, except that coverage continues for a grace period of 31 days after the premium due date. The Applicant may be responsible to pay premium for the grace period coverage. If before any premium due date the Applicant provides advance written notice to the Insurer of request to cancel, then the grace period coverage does not apply. In addition to reasons for termination that are specified in the group insurance Policy, the Insurer may also reform or rescind for fraud or material misrepresentation. The Insurer will provide the Applicant with a minimum of 31 days advance written notice of termination date unless due to nonpayment of premium, fraud or misrepresentation ; . Termination will not prejudice a valid claim existing on the termination date, unless due to nonpayment of premium, fraud or misrepresentation. Upon termination, Applicant may request reinstatement of coverage by paying all applicable premium, plus a nonrefundable reinstatement fee when allowed by state law. Insurer will deposit payment during review of Applicant's request. Depositing Applicant's check does not mean acceptance and does not guarantee reinstatement. Insurer can approve or decline reinstatement request and will notify Applicant in writing and aromasin. The Patient Advocate Foundation 1-800-532-5274 700 Thimble Shoals Blvd., Suite 200, Newport News VA 23606 patientadvocate Provides education, legal counseling, and referrals to cancer patients and survivors concerning managed care, insurance, financial issues, job discrimination, and debt crisis matters. The Patient Advocate Foundation also provides co-payment assistance for pharmaceutical products to insured Americans who qualify for their Patient Assistance Program. Rx Assist 1-800-877-8442 volunteersinhealthcare Database of patient assistance programs offered by pharmaceutical companies. Senior HICAP Services 301 W. Preston Street, Suite 1007, Baltimore, MD 21201-2374 410-767-1100 Program that enlists health insurance counselors to assist seniors with questions regarding Medicare or Medicaid. Also provides information on long-term care insurance. Call for the HICAP number in your county. Shirley B. Robinson Memorial Fund P.O. Box 116, Smithburg, MD 21783 Jessie Kretzer - 301-824-3030 shirleyslegacy Financial assistance for cancer patients and their families in Washington County and surrounding areas. Social Security Adminstration SSA ; 1-800-772-1213, TTY: 1-800-325-0778 : ssa.gov Provides a monthly income for eligible elderly and disabled individuals. Information on eligibility, coverage, and how to apply for benefits is available from the Social Security Administration. Substrates, modifying in this way signal transduction or regulatory pathways Daigle et al., 1999 ; . M. genitalium has been proposed as a suitable model to achieve an in-depth understanding of the biology of a freeliving organism Roberts, 2004 ; . Fulfilling this goal depends on the development of new genetic tools and, in particular, on the identification of new selectable markers for M. genitalium genetic studies. In this work we describe the construction of a modified tetM gene tetM438 ; that can be used in M. genitalium. Comparison between the tetM438 and aac 69 ; -aph 20 ; genes clearly shows a negative effect of aac 69 ; -aph 20 ; on the growth of M. genitalium. Moreover, the use of tetM438 increases dramatically the number of transformants obtained and reduces considerably the time needed for colonies to become visible. Using tetM438 in conjunction with a minitransposon derived from Tn4001 MTn4001 ; , we have been able to obtain insertions in genes which were previously considered to be necessary for M. genitalium growth in laboratory conditions Hutchison et al., 1999. UISPP IUPPS XV Congress XV Congrs Book of Abstracts Livre des Rsums C52-04 `In the eye of the beholder': a re-evaluation of passage tombs in the Irish Neolithic landscape through the integration of spatial, visibility and archaeoastronomical data. Massimo CULTRARO Italy ; Geometric motions on vessel and hidden symmetries into community: a study of the painted pottery from EBA Sicily Italy ; in reconstructing patterns of social memory. Emilia PASZTOR Szazhalombatta, Hungary ; The significance of the sun, moon and celestial bodies to societies in the Carpathian Basin during the Bronze Age. Michael RAPPENGLECK Munich, Germany ; The "domestication" of the world into a house and a home: Cosmographic symbolism as a basic expression of the human mind. Andrea VIANELLO Oxford, UK ; The ship and its symbolism in European prehistory. Fernando COIMBRA Salamanca, Spain ; Cognitive archaeology, rock art and achaeoastronomy: interrelated disciplines. George DIMITRIADIS Philippi, Greece ; Space Analysis as Cognitive Approach to Prehistoric Mentality. SUSHENG Beijing, China ; Howl cognitive mask in Rock Art. Davide DELFINO Milano, Italy ; Simbologie du Metal: le cas des depots dans les Alpes de l'Ouest au Bronze Final. Romeo DUMITRESCU Bucharest, Romania ; Fertility kits. A possible approach of the female representations in Precucuteni Cucuteni cultures. Dragos GHEORGHIU Bucharest, Romania ; Symbolic technologies in Chalcolithic Clay Cultures. Li WINTER Stockholm, Sweden ; From Diffusion to Interaction: connections between the Nordic Area and the Mediterranean Cultures during the 2nd Millennium BC.
Women's health: antispasmodic action of chamomile soothes the menstrual cramps and to lessen the possibility of premature labor. The pathobiological processes involved in causing fostering diabetes, and has designed therapeutic agents with a range of action to fight hyperglycemia, the efficacy of these therapeutic agents is compromised in several ways. For individual agents act only on part of the pathogenic process and only to a partial extent1821. This may be the reason that even after so much advancement in understanding the disease process and availability of a wide range of therapeutic agents, the disease is still progressing. Multiple defects in the pathophysiology of diabetes are mostly imprecisely understood, and therefore warrant not isolating a single drug target to the reversal of all or majority of aspects of the disease17, as biological systems are too complex to be fully understood through conventional experimentation and also because they are nonlinear. They also may have properties that are not obvious from biological considerations alone. For example, though hyperglycemia is a classical risk factor for the development of diabetic complications, there is no consensus regarding the pathogenic links between hyperglycemia and diabetic complications22. There are a number of equally tenable hypotheses on the origin of complications beyond hyperglycemic consideration. Therefore, the unidirectional therapeutic approach in the management of diabetes does not appear to be the way to address this problem. On the other hand, the therapeutic approach of several traditional medicinal systems is more holistic. The fundamental mechanisms of these medicinal systems are still unexplainable using modern tools. The medicinal preparations in traditional medicines contain a variety of herbal and non-herbal ingredients that are thought to act on a variety of targets by various modes and mechanisms. This article presents an overview of diabetic pathogenesis, particularly impaired carbohydrate metabolism leading to hyperglycemia, diabetes and oxidative stress. It analyses how herbal medicines and their ingredients correct manipulate the vitiated homeostasis of carbohydrate metabolism providing multiple therapeutic benefits in diabetes mellitus, and also discusses the present status and future prospects of these medicines. against hypoglycemia during periods of fasting and against excessively high levels following the ingestion of a high carbohydrate diet. These goals are met chiefly through the hormonal modulation of the production of glucose by the liver and the peripheral uptake of glucose by skeletal muscle, heart muscle and fat. When mammals fast, glucose homeostasis is achieved by triggering expression of gluconeogenic genes in response to glucagon, and when they take a carbohydrate-rich diet, the function is taken over by insulin for its uptake and utilization peripherally. The impairment in glucose metabolism, therefore, may lead to physiological imbalance and warrants proper management. Starting from the carbohydrate ingestion, breakdown into the monosaccharides, their building blocks glucose, sucrose and fructose to the downhill utilization of glucose for generation of energy, Figure 1 presents in brief a systematic sketch of the pathways involved in carbohydrate metabolism. Any vitiation, therefore, in normal glucose metabolic pathway may lead to the impaired glucose metabolism, the onset of hyperglycemia and subsequently, diabetes mellitus. This flow chart also presents points where modern therapeutics targets and traditional medicines have shown their therapeutic potential. After the breakdown of carbohydrates by digestive enzymes, the released glucose becomes the primary stimulus for the -cells of the pancreatic islets. Prolonged exposure of pancreatic islets to elevated glucose concentration has been shown both in vitro and in vivo to impair glucose-stimulated insulin release23, 24. Glucose stimulation of pancreatic islet -cells initiates a cascade of events resulting in insulin secretion and is dependent on an increase in intracellular Ca2 + refs 25 and 26 ; . This increases the phosphoinositide hydrolysis, inositol 1, 4, 5triphosphate IP-3 ; production and mobilizes Ca2 + from intracellular IP-3-sensitive Ca2 + stores in the pancreatic -cells27. Regulation of glucose metabolism is a key aspect of metabolic homeostasis and insulin is the dominant hormone influencing this regulatory system. One of the major effects of insulin is to enhance overall glucose disposal, and this is achieved by stimulation of glucose uptake into the target tissues. This task is facilitated by insulin-sensitive glucose transporter GLUT-4 ; , which is uniquely expressed in skeletal muscles, cardiac muscles and adipose tissues28. This action of insulin in the regulation of glucose homeostasis in post-absorptive state is a very important function in maintaining euglycemia and preventing hyperglycemia29. The molecular and cellular biology of GLUT-4 is a complicated science. Further, understanding of GLUT-4 biology may provide novel therapeutics in insulin resistance in Type 2 diabetes, for GLUT-4 is the key facilitator responsible for the maintenance of euglycemia in the body30. Glucose-stimulated release of insulin and insulin-guided metabolism of glucose are therefore, the primary balancing factor in maintaining euglycemic state in the blood Figure 1 ; . How31. Add a rating - post your opinion about this drug post your opinion about antispasmodic 1-4 of 9 next page - conditions of use: the information in this database is intended to supplement, not substitute for, the expertise and judgement of healthcare professionals.
Trachea and larynx by flow-volume loops. Rev Respir Dis 1973; 108: 475-81 Evans MA, Triggs EJ, Broe GA, Saines N. Systemic availability of orally administered L-dopa in the elderly parkinsonian patient. Eur J Clin Pharmacol 1980; 17: 215-21 Rossor MN, Watkins J, Brown MJ, Reid JL, Dollery CT. Plasma levodopa, dopamine and therapeutic response following levodope therapy ofparkinsonian patients. J Neurol Sd 1980; 46: 38592 Quinn NP Anti-parkinsonian drugs today. Drugs 1984; 28: 23662 Spokes EGS, Bannister R, Oppenheimer DR. Multiple system atrophy with autonomic failure. J Neurol Sd 1979; 43: 59-82 Guindi GM, Bannister R, Gibson WPR, Payne JK. Laryngeal electromyography in multiple system atrophy with autonomic failure. J Neurol Neurosurg Psychiatry 1981; 44: 49-53 Nugent CA, Harris HW, Cohn J, Smith CC, Tyler FH. Dyspnea as a symptom in Parkinson's syndrome. Rev Tuberc 1958. Buy cheap antispasmodicBuy antispasmodic onlineWhile Gesualdo's church music shows more self-discipline and restraint than his madrigals, which comprised the great bulk of his output, it came late in his career and by then his welldeveloped musical personality could not be suppressed. Whenever the music isn't stunning you with its sensual beauty, it's crackling with excitement and dramatic intensity. Gesualdo published two books of motets in 1603, followed by his settings for Tenebrae in 1611, some of which are featured in this new recording by the Taverner Consort and Choir. He often chose texts of a penitential character. "Tenebrae, " meaning shadows or darkness, refers to an office sung during Holy Week. Gesualdo composed settings for Tenebrae on Maundy Thursday, Good Friday, and Holy Saturday. This recording features his nine Good Friday motets, placed in their liturgical context by the inclusion of most of the plainsong2 propers. Gesualdo's style is perfectly captured in this newly released recording by the Taverner Consort and Choir. The consort, which sings all of Gesualdo's motets here, consists of seven male voices three altos and tenors and one bass ; . The choir sings antiphonally with the consort in the psalms and other plainsong propers. This is plainsong singing of the highest order it has a very monastic flavor but the singers are highly trained professionals whose sense of ensemble is remarkable. And in Gesualdo's motets, which are interspersed throughout, the Taverner Consort combines great virtuosity and pitch accuracy with silky sensuality achieved though subtle crescendi and diminuendi ; and pointedly dramatic delivery, according to the demands of the music. I can't imagine a more perfectly realized account. It's superior to the more refined approach of the Tallis Scholars, which makes the music sound too much like Palestrina, and even to the stylish and dramatic singing of Les Arts Florissants in their excellent recording of Gesualdo's madrigals [Gesualdo: Madrigaux, Les Arts Florissants, Harmonia Mundi CD 901268]. Somehow Parrott and the Taverner group find a balance between these extremes that serves the music well. While not as vivid as the very best, this recording is quite successful sonically. It was recorded in St. Bar-tholomew's Church, London, England, evidently a fairly resonant venue. It's miked a bit. Bacterias anaerobias esporuladas. In: Farreras, Rozman. Medicina Interna. 14th Ed. Madrid: Harcourt; 2000; II: 2632-2640. Caya JG. Clostridium botulinum and the ophthalmologist: a review of botulism, including biological warfare ramifications of botulinum toxin. Surv Ophthalmol 2001; 46: 25-34. Savino PJ, Sergott RC. Neurooftalmologa. In: Rhee, Pyfer. Manual de Urgencias Oftalmolgicas. 3rd Ed. Philadelphia: Mc Graw-Hill Interamericana; 2001; 301-364. Penas SC, Faria OM, Serrao R, Capao-Filipe JA, MotaMiranda A, Falcao-Reis F. Ophthalmic Manifestations in 18 patients with botulism diagnosed in Porto, Portugal between 1998 and 2003. J Neuroophthalmol 2005; 25: 262-267. Simcock PR, Kelleher S, Dunne JA. Neuro-ophthalmic findings in botulism type B. Eye 1994; 8: 646-648. Order antispasmodic online |
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