|
401 Jones Rd. Oceanside, CA 92054 1216 Contact: Serenity Gardner, Ph : 760-901-2529 Fax: 760-901-2578 Email: smccoon hydranautics Web: : membranes Product Service Description: Hydranautics manufactures reverse osmosis and ultrafiltration membranes for an array of water treatment applications which include: potable water, ultrapure water, waste treatment, and seawater desalination.
Conclusions Our study failed to detect a beneficial effect of early or more potent anti toxoplasma treatment on the risks of intracranial or ocular lesions in children with congenital toxoplasmosis. However, larger, prospective studies, which determine the effect of prenatal treatment on long-term developmental outcomes are required to justify changes in clinical practice. Keywords Accepted Congenital toxoplasmosis, prenatal treatment, intracranial calcification, retinochoroiditis 24 May 2001.
Table 14. Number and percent distribution of injury-related emergency department visits with corresponding standard errors, by intent and mechanism of external cause: United States, 2005.
As cancer diagnosis and treatment depends increasingly on the use of molecular markers for assessment of prognosis, treatment selection and agent development, this meeting has become an important venue for discussion of contemporary issues in molecular pathology, assay development, clinical trial design and the evolution of promising serum and tissue markers from research tools to clinical useful laboratory tests. Conference participants include clinicians, pathologists, laboratory scientists, statisticians and representatives of industry and regulatory agencies.
Wyeth comments on documents received 31st August 2006 3. Patients who failed one anti-TNF and switched to another a. A subgroup was identified with 6 months on the 2nd drug The unadjusted HAQ change data was: Group 1 Group 2 Group 3 Group 3a -0.07 -0.01 -0.12 -0.15.
It has developed and has on file, has not developed and does not have on file, at each establishment, affirmative action programs required by rules and regulations of the Secretary of Labor 41 CFR Subparts 60-1 and 60-2 ; , or has not previously had contracts subject to the written affirmative action programs ii ; It requirement of the rules and regulations of the Secretary of Labor. e ; Certification Regarding Payments to Influence Federal Transactions 31 U.S.C. 1352 ; . Applies only if the contract is expected to exceed 0, 000. ; By submission of its offer, the offeror certifies to the best of its knowledge and belief that no Federal appropriated funds have been paid or will be paid to any person for influencing or attempting to influence an officer or employee of any agency, a Member of Congress, an officer or employee of Congress or an employee of a Member of Congress on his or her behalf in connection with the award of any resultant contract. f ; Buy American Act Certificate. The certificate at DFARS 252.225-7000 or 7020 shall be completed if it is provided as an Attachment to FAR 52.212-3 ; . g ; Buy American Act - North American Free Trade Agreements Israeli Trade Act Certificate, Alternates I and II Trade Agreements Certificate. The certificate in DFARS 252.225-7035 shall be completed if it is provided as an Attachment to 52.212-3. ; h ; Certification Regarding Debarment, Suspension or Ineligibility for Award Executive Order 12689 ; . Applies only if the contract value is expected to exceed the simplified acquisition threshold. ; The offeror certifies, to the best of its knowledge and belief, that the offeror and or any of its principals-Are, are not presently debarred, suspended, proposed for debarment, or declared ineligible 1 ; for the award of contracts by any Federal agency; Have, have not, within the three-year period preceding this offer, been convicted of or had a 2 ; civil judgment rendered against them for: commission of fraud or a criminal offense in connection with obtaining, attempting to obtain, or performing a Federal, state or local government contract or subcontract; violation of Federal or state antitrust statutes relating to the submission of offers; or commission of embezzlement, theft, forgery, bribery, falsification or destruction of records, making false statements, tax evasion, or receiving stolen property; and Are, are not presently indicted for, or otherwise criminally or civilly charged by a 3 ; Government entity with, commission of any of these offenses and peppermint.
In Table 1. The average age was 42 yr, and the female to male ratio was 5.2: 1. Thyroid size averaged approximately 50 g 2.5 times normal ; , and the average 24-h 123I uptake was 58%. Before treatment, 109 patients 43% ; received PTU, and 98 39% ; had received methimazole. Clinical Graves' ophthalmopathy was noted in 23% of patients; the vast majority were noted to be mild, consisting of exophthalmus and or conjunctival irritation. All patients had stopped antithyroid drug ATD ; therapy before 131I therapy, and most patients 83% ; were off ATD for 57 d before 131I treatment. All other patients were off ATD for greater than 7 d. The average dose of 131I was 14.6 mCi 540 MBq ; , resulting in an estimated retained dose of 8.1 mCi 300 MBq ; 131I in the thyroid at 24 h. The average 131I retained in the thyroid per estimated g of tissue at 24 h was 173 Ci g [6.4 MBq; range, 50 450 Ci g 1.8 16.6 MBq ; ]. At 1 yr, 225 patients 86% ; were successfully treated hypothyroid or euthyroid ; , and 36 14% ; remained hyperthyroid. Among the former group, 217 had persistent hypothyroidism 83% ; , and 8 3% ; were euthyroid. Among the hypothyroid group, 31 patients 12% ; had persistent hyperthyroidism, and 5 2% ; had hypothyroidism, followed by recurrent hyperthyroidism. Of those successfully treated, over 90% responded to 131I therapy within the first 6 months, as judged by biochemical analysis, symptom improvement, and or decreasing requirements for antithyroid medications; the remainder responded within the first year. There was a significant correlation between estimated thyroid weight and 24-h 123I uptake P 0.01; data not shown ; . As expected because the estimated thyroid weight was not included in dose calculations, there was an inverse correlation between thyroid size and the dose of 131I present at 24 h thyroid tissue P 0.01 ; . The success of treatment was directly related to the dose of 131I retained per estimated g of thyroid tissue, but this relationship was not linear Fig. 1 ; . No patient became hypothyroid if the estimated 24-h dose was less than 80 Ci 3.0 MBq ; g The failure rate decreased progressively to reach approximately 10% at 128 155 Ci 4.75.7 MBq ; g. It did not decrease appreciably below that despite doses up to 400 Ci 14.8 MBq ; g thyroid at 24 h Fig. 1.
Habits, medication, and level of physical activity or exercise also helps the doctor determine the cause of constipation and percodan.
If r is removable protecting group then it leads to the synthesis of pentostatin aglyconean intermediate suitable for glycosylation to give pentostatin 1a.
Stuecklschweiger, G., Arian-Schad, K. S., Kapp, D. S., Handl-Zeller, L., & Hackl, A. G. 1993, "Analysis of temperature distributions of interstitial hyperthermia using a hot water system", International Journal of Radiation Oncology Biology Physics, vol. 26, no. 5, pp. 891-895. Reason for exclusion: Title abstract first pass ; : Excluded. Sudarshan, G. & Crawford, D. 1992, "Anaesthesia for intraperitoneal hyperthermic perfusion", Anaesthesia, vol. 47, no. 6, pp. 483-485. Reason for exclusion: Title abstract first pass ; : Excluded. Suga, K., Fujita, T., Nakada, T., Yoneshiro, S., Uchisako, H., Nishigauchi, K., Nakanishi, T., & Hamada, Y. 1993, "Preliminary Tl-201 SPECT for assessment of treatment efficacy in three patients with pancreatic cancer", Clinical Nuclear Medicine, vol. 18, no. 9, pp. 771-775. Reason for exclusion: Title abstract first pass ; : Excluded. Sugarbaker, P. H. & Steves, M. A. 1993, "A cytoreductive approach to treatment of multiple liver metastases", Journal of Surgical Oncology, vol. 53, no. SUPPL. 3, pp. 161-165. Reason for exclusion: Title abstract first pass ; : Excluded. Sugarbaker, P. H., Averbach, A. M., Jacquet, P., Stephens, A. D., & Stuart, O. A. 1996, "A simplified approach to hyperthermic intraoperative intraperitoneal chemotherapy HIIC ; using a self retaining retractor", Cancer treatment and research, vol. 82, no. -, pp. 415-421. Reason for exclusion: Title abstract first pass ; : Included. Title abstract second pass ; : Included. Title abstract third pass ; : Excluded. Sugarbaker, P. H., Sugarbaker, C., Stephens, A. D., & Chang, D. 2000, "Radiofrequency hyperthermia in the palliative treatment of mucinous carcinomatosis of appendiceal origin: Optimizing and monitoring heat delivery in western patients", International Journal of Hyperthermia, vol. 16, no. 5, pp. 429-441. Reason for exclusion: Title abstract first pass ; : Included. Title abstract second pass ; : Included. Title abstract third pass ; : Included. Full paper: Excluded. Not microwave hyperthermia. Sugihara, M., Fujita, Y., Enomoto, K. I., Maeno, T., & Ishida, T. 1994, "Induction of differentiation by radiation and hyperthermia in neuroblastoma-glioma hybrid cells", Cell Biochemistry and Function, vol. 12, no. 2, pp. 137-142. Reason for exclusion: Title abstract first pass ; : Excluded. Sugimachi, K., Inokuchi, K., & Kai, H. 1984, "Preoperative hyperthermo-chemoradiotherapy effective for carcinoma of the esophagus", Journal of Surgical Oncology, vol. 27, no. 3, pp. 199-204. Reason for exclusion: Title abstract first pass ; : Included. Title abstract second pass ; : Excluded. Sugimachi, K., Kai, H., & Inokuchi, K. 1985, "5. Preoperative hyperthermo-chemoradiotherapy for esophageal carcinoma. Analysis of 20 cases", Japanese journal of medicine, vol. 24, no. 1, pp. 80-83. Reason for exclusion: Title abstract first pass ; : Included. Title abstract second pass ; : Excluded and pergolide.
Tested HCL patients who were previously enrolled on the phase I trial BL14 and BL41 ; or who were enrolled by special exemption before phase II testing BL47 and BL49 ; . Patient BL14 Fig. 4A ; , who previously had CR to BL22, had slowly decreasing disease burden during cycles 19 to 31 given several years later after relapse. During this time, the disease by RQPCR decreased from 2, 100 F 580 to 4.5 F 1.3, a change of 2.7 logs over nearly 14 months. Disease by flow cytometry during the same interval decreased 2.1 logs from 3, 500 to 29 cells AL. Despite resistance to rituximab and pentostatin when administered separately before cycle 1 of BL22, this patient had CR to the rituximab pentostatin combination 59 months after beginning BL22. MRD was undetectable in the blood by flow.
Two things stand out here. Firstly, environmental protection has evidently become a political matter for top management in many industrial countries. Secondly, awards are not being presented for environmental protection facilities and processes, but for endeavors made by companies to develop products that have no impact on the environment and, in ideal circumstances, also protect it from any adverse effects. Both of these apply to us. At Bayer, environmental protection is a matter for the top echelons of management. Having said that, every individual employee is as stated in our Guidelines for Responsible Care, Health Protection and Safety also com and permax.
19 17. Kaya, T., T. Utkan, Y. Sarioglu, and M. Goksel. Altered endothelium-mediated relaxation by sympathectomy in isolated rabbit carotid artery rings. Methods Find. Exp. Clin. Pharmacol. 17: 369-375, 1995. Komori, K., K. Takeuchi, S. Ohta, S. Funahashi, M. Ishida, T. Matsumoto, T. Onohara, M. Kume, and K. Sugimachi. Properties of endothelium and smooth muscle cells in canine femoral arteries after lumbar sympathectomy. Eur. J. Surg. 165: 1086-1090, 1999. Lepori, M., C. Sartori, H. Duplain, P. Nicod, and U. Scherrer. Sympathectomy potentiates the vasoconstrictor response to nitric oxide synthase inhibition in humans. Cardiovasc. Res. 43: 739-743, 1999. Lindqvist, M., S. Davidsson, P. Hjemdahl, and A. Melcher A. Sustained forearm vasodilation in humans during mental stress is not neurogenically mediated. Acta Physiol. Scand. 158: 7-14, 1996. Lowell, R.C., P. Gloviczki, K.J. Cherry Jr., T.C. Bower, J.W. Hallett Jr., A. Schirger, and P.C. Pairolero. Cervicothoracic sympathectomy for Raynaud's syndrome. Int. Angiol. 12: 168-172, 1993. Murray, J.G., and J.W. Thompson. Collateral sprouting in response to injury of the autonomic nervous system, and its consequences. Br. Med. Bull. 13: 213219, 1957. Nicholson, M.L., M.J. Dennis, and B.R. Hopkinson. Endoscopic transthoracic sympathectomy: successful in hyperhidrosis but can the indications be extended? Ann. R. Coll. Surg. Engl. 76: 311-314, 1995.
Commissioning strategy is to leave nuclear plants in a SAFSTOR condition for up to 100 years, Woollam said. This strategy is driven primarily by the lack of disposal facilities for waste and by a lack of funding. However, he stated, this strategy is not popular with the public nor with regulators. The U.K.'s new Nuclear Decommissioning Authority is preparing a new strategy document, which is due to go to the government in December. It is "highly likely, " Woollam said, that this strategy will recommend a different way to deal with the old Magnox sites, possibly recommending that they be decommissioned within 25 years instead of 100 years. There are 26 Magnox plants in the United Kingdom, Woollam said, and they are "huge" compared with pressurized water reactors, with likewise huge amounts of decommissioning waste. This material is not highly radioactive, he said, but it is contaminated, and there is nowhere in the United Kingdom to put this material at the moment. David Reed, from British Nuclear Group, pointed out that the Sellafield site, located in the Lake District in northwest England, makes up about 60 to 70 percent of the country's civil nuclear liability. The 2-square-mile site, location of an ordnance factory during World War II, holds 1000 buildings having a great age range. Among the facilities at the site is the Calder Hall power and perphenazine.
1. Myint H, Copplestone JA, Orchard J, et al. Fludarabinerelated autoimmune haemolytic anaemia in patients with chronic lymphocytic leukaemia. Br J Haematol. 1995; 91: 341344. Weiss RB, Freiman J, Kweder SL, Diehl LF, Byrd JC. Hemolytic anemia after fludarabine therapy for chronic lymphocytic leukemia. J Clin Oncol. 1998; 16: 1885-1889. Fleischman RA, Croy D. Acute onset of severe autoimmune hemolytic anemia after treatment with 2chlorodeoxyadenosine for chronic lymphocytic leukemia. J Hematol. 1995; 48: 293. Aslan DL, Peterson BA, Long-Tsai M, Eastlund T. Earlyonset autoimmune hemolytic anemia after cladribine therapy for Waldenstrom's macroglobulinemia. Transfusion. 2006; 46: 90-94. Byrd JC, Hertler AA, Weiss RB, Freiman J, Kweder SL, Diehl LF. Fatal recurrence of autoimmune hemolytic anemia following pentostatin therapy in a patient with a history of fludarabine-associated hemolytic anemia. Ann Oncol. 1995; 6: 300-301. Paydas S. Fludarabine-induced hemolytic anemia: successful treatment by rituximab. Hematol J. 2004; 5: 81-83. Young PP, Uzieblo A, Trulock E, Lublin DM, Goodnough LT. Autoantibody formation after alloimmunization: are blood transfusions a risk factor for autoimmune hemolytic anemia? Transfusion. 2004; 44: 67-72. Garratty G. Autoantibodies induced by blood transfusion.
203 Multiple myeloma and immunoproliferative neoplasms The following fifth-digit subclassification is for use with category 203: 0 without mention of remission 1 in remission 203.0 Excludes: 203.1 203.8 Multiple myeloma Kahler's disease Myelomatosis solitary myeloma 238.6 ; Plasma cell leukemia Plasmacytic leukemia Other immunoproliferative neoplasms and phenazopyridine.
Typical antipsychotics are far more active on D2 receptors than atypical antipsychotics. However, D2 receptors predominate in the basial ganglia, and a decrease in dopamine will cause Parkinson-type symptoms, which are referred to as extrapyramidal side effects. They are divided into three drug groups, which are listed below. They basically have a similar mode of action and their use is based upon the doctor's preference and or the patient's tolerance. This is usually a case of trial and error and pentostatin.
Fore apply to the target population of these recommendations. Our findings concur with the recommendations of the joint panel of the American College of Physicians-American Society of Internal Medicine and the American College of Chest Physicians, which recommended against use of methylxanthines for exacerbations of chronic obstructive pulmonary disease.38 Limitations The major limitation of this meta-analysis was the paucity of randomised trial data. The sparseness of the data prevented the assignment of definitive conclusions regarding benefits of methylxanthines but allowed firmer conclusions regarding their effect on adverse events. There is a possibility of publication and selection bias in any meta-analysis; but publication bias is unlikely to affect this analysis since the published trials were predominantly negative. The data were not evaluated for the presence of publication bias since too few trials were available to perform a meaningful evaluation. To avoid selection bias, a systematic and comprehensive search was conducted and two reviewers independently evaluated trials for inclusion. The available data do not support the use of methylxanthines to treat exacerbations of chronic obstructive pulmonary disease. Potential benefits of methylxanthines on lung function and symptoms were generally not confirmed at standard levels of and phenelzine.
2003 ; , etorphine caused AC superactivation to a much higher level than levorphanol. Our results are similar to the findings that DAMGO, but not morphine, caused phosphorylation and internalization of the opioid receptor without overexpression of GRK2 for example, Keith et al., 1996; Zhang et al., 1998 ; , but the two drugs induced similar levels of AC superactivation Blake et al., 1997a ; . However, our observations are different from those of Finn and Whistler 2001 ; , who, using mutant receptors and various agonists, showed that lack of internalization of the opioid receptor resulted in higher degrees of AC superactivation. The reasons for the differences are not clear. AC Superactivation after Prolonged Activation of the Opioid Receptor. Our results on AC superactivation after U50, 488H pretreatment of the human or rat receptor are similar to those of Avidor-Reiss et al. 1995b ; , who reported that incubation of CHO cells stably transfected with the rat opioid receptor with 1 M U69, 593 for 4 h resulted in 250% enhancement of AC activity. In addition, we found that AC superactivation was U50, 488H concentration-dependent with an EC50 value of about 25 nM, which was about 6 times higher than that for stimulation of [35S]GTP S binding Zhu et al., 1997 ; . Avidor-Reiss et al. 1995b ; also found that the EC50 value of U69, 593 for inducing AC superactivation was much higher than that for inhibiting AC. We demonstrated U50, 488H 1.5 M ; -induced AC superactivation was time-dependent, occurring as early as 1 h and with t1 2 of Such a rapid time course suggests that protein synthesis is not required for AC superactivation. Mechanisms of AC Superactivation. In addition to and opioid receptors, AC superactivation has been observed for several other Gi o-coupled receptors after prolonged agonist treatment for a review, see Watts, 2002 ; . The molecular mechanisms leading to AC superactivation have not been fully elucidated. A model was recently proposed by Watts 2002 ; , in which the withdrawal of drugs after chronic stimulation led to the dissociation of G i from G , which in turn, induces AC superactivation in a G s-dependent manner. AC superactivation after chronic opioid treatment has been shown to be mediated by Gi o proteins Avidor-Reiss et al., 1995a ; . Each Gi subunit may play a role Tso and Wong, 2000, 2001 ; and G subunits play an essential role AvidorReiss et al., 1996 ; . Prolonged opioid treatment decreased the detergent solubility, and presumably lipid rafts microdomain localization, of G i and 1 subunits, which coincided with AC superactivation Bayewitch et al., 2000 ; . Chronic opioid treatment resulted in an accumulation of the depalmitoylated form of Gs in the plasma membrane, which is associated with AC superactivation Ammer and Schulz, 1997 ; . AC superactivation required Gs -mediated AC stimulation, but it was not a direct Gs effect; rather, it involved a secondary regulatory event that requires stimulation of AC by Ammer and Schulz, 1998 ; . Prolonged opioid receptor activation induces superactivation of AC types I, V, VI, and VIII, but not of type II, III, IV, or VII, expressed in CHO cells Avidor-Reiss et al., 1996, 1997 ; . Chronic morphine administration in vivo resulted in up-regulation and enhanced protein-kinase C-mediated phosphorylation of the AC II isoforms in guinea pig longitudinal muscle myenteric plexus, leading to a shift in opioid receptor G-protein signaling from predominantly Gi inhibitory to G stimulatory effect on.
VIRGINIA-Richmond Area. Retiring dentist in new professional building new equipment four operatories. Modern established computerized practice for immediate sell. Contact: Box 1205, Chester, 23831 and phenobarbital.
Pentostatin no prescription
At Six Flags Great Adventure in Jackson, N.J., children share a group hug with Looney Tunes character Daffy Duck and peppermint.
17. Albert CM, McGovern BA, Newell JB, Ruskin JN. Sex differences in cardiac arrest survivors. Circulation. 1996; 93: 1170 Doval HC, Nul DR, Grancelli HO, Varini SD, Soifer S, Corrado G, Dubner S, Scapin O, Perrone SV, GESICA-GEMA Investigators. Nonsustained ventricular tachycardia in severe heart failure: Independent marker of increased mortality due to sudden death. Circulation. 1996; 94: 3198 Escobedo LG, Zack MM. Comparison of sudden and nonsudden coronary deaths in the United States. Circulation. 1996; 93: 20332036. Leor J, Poole WK, Kloner RA. Sudden cardiac death triggered by an earthquake. N Engl J Med. 1996; 334: 413 Brugada P, Talajic M, Smeets J, Mulleneers R, Wellens HJ. The value of the clinical history to assess prognosis of patients with ventricular tachycardia or ventricular fibrillation after myocardial infarction. Eur Heart J. 1989; 10: 747752. Friedlander Y, Siscovick DS, Weinmann S, Austin MA, Psaty BM, Lemaitre RN, Arbogast P, Raghunathan TE, Cobb LA. Family history as a risk factor for primary cardiac arrest. Circulation. 1998; 97: 155160. Stevenson WG, Stevenson LW, Middlekauff HR, Saxon LA. Sudden death prevention in patients with advanced ventricular dysfunction. Circulation. 1993; 88: 29532961. Weinberg BA, Miles WM, Klein LS, Bolander JE, Dusman RE, Stanton MS, Heger JJ, Langefeld C, Zipes DP. Five-year follow-up of 589 patients treated with amiodarone. Heart J. 1993; 125: 109 Herre JM, Sauve MJ, Malone P, Griffin JC, Helmy I, Langberg JJ, Goldberg H, Scheinman MM. Long-term results of amiodarone therapy in patients with recurrent sustained ventricular tachycardia or ventricular fibrillation. J Coll Cardiol. 1989; 13: 442 Bardy GH, SCD-HeFT Investigators. Prevention of sudden cardiac death in patients with congestive heart failure. In: Woosley RL, Singh SN, eds. Clinical Trials on the Treatment of Arrhythmia. New York, NY: Marcel Decker, Inc. In press. 27. Zabel M, Klingenheben T, Franz MR, Hohnloser SH. Assessment of QT dispersion for prediction of mortality or arrhythmic events after myocardial infarction: results of a prospective, long-term follow-up study. Circulation. 1998; 97: 25432550. Zipes DP. Unwitting exposure to risk. Cardiol Rev. 1993; 1: 13. Moss AJ, Hall WJ, Cannom DS, Daubert JP, Higgins SL, Klein H, Levine JH, Saksena S, Waldo AL, Wilber D, Brown MW, Heo M, Multicenter Automatic Defibrillator Implantation Trial Investigators. Improved survival with an implanted defibrillator in patients with coronary disease at high risk for ventricular arrhythmia. N Engl J Med. 1996; 335: 19331940. Fuster V, Poon M, Willerson JT. Learning from the transgenic mouse: endothelium, adhesive molecules, and neointimal formation. Circulation. 1998; 97: 16 Tomaselli GF, Beuckelmann DJ, Calkins HG, Berger RD, Kessler PD, Lawrence JH, Kass D, Feldman AM, Marban E. Sudden cardiac death in heart failure: the role of abnormal repolarization. Circulation. 1994; 90: 2534 Kaab S, Nuss HB, Chiamvimonvat N, O'Rourke B, Pak PH, Kass DA, Marban E, Tomaselli GF. Ionic mechanism of action potential prolongation in ventricular myocytes from dogs with pacing-induced heart failure. Circ Res. 1996; 78: 262273. Mitrani RD, Klein LS, Miles WM, Hackett FK, Burt RW, Wellman HN, Zipes DP. Regional cardiac sympathetic denervation in patients with ventricular tachycardia in the absence of coronary artery disease. J Coll Cardiol. 1993; 22: 1344 Calkins H, Allman K, Bolling S, Kirsch M, Wieland D, Morady F, Schwaiger M. Correlation between scintigraphic evidence of regional sympathetic neuronal dysfunction and ventricular refractoriness in the human heart. Circulation. 1993; 88: 172179. Muller JE, Kaufmann PG, Luepker RV, Weisfeldt ML, Deedwania PC, Willerson JT, for the Mechanisms Precipitating Acute Cardiac Events Participants. Mechanisms precipitating acute cardiac events: review and recommendations of an NHLBI workshop. Circulation. 1997; 96: 32333239. Peckova M, Fahrenbach CE, Cobb LA, Hallstrom AP. Circadian variations in the occurrence of cardiac arrests: initial and repeat episodes. Circulation. 1998; 98: 3139. Schwartz PJ, Priori SG, Napolitano C. Long QT syndrome. In: Zipes DP, Jalife J, eds. Cardiac Electrophysiology: From Cell to Bedside. Orlando, Fla: WB Saunders. 1999. In press and phenylephrine.
|